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  Home > Canine Supplements >

  Animal Naturals- K9 Puppy Gold
  Milk replacement for puppies, newborn puppies, puppy supplement
Give your puppies a healthy start.
 
We will not be undersold.  Email sales@healthyplanetrx.com if you see Animal Naturals products advertised at a lower price.


K9 Puppy Gold helps every puppy be the pick of the litter.  Puppy Gold is also great for nursing mothers and hard gainers.  This is the next best thing to mother's milk.  It doesn't get any better than Puppy Gold! Animal Naturals Puppy Gold is 100% human grade and developed to enhance the life of your small canine companion.  K9 Gold is also great for debilitated dogs and/or post surgery dogs.  Puppy Gold promotes good canine health and is one of our finest dog healthy supplments for puppies and nursing mothers.  FREE SHIPPING ON 15 and 32 LB SIZE.



Item# Item Name List Price Our Price Qty Add
AN-PG-15 Animal Naturals- K9 Puppy Gold- 15lb Container (FREE SHIPPING) $164.99 $143.54
AN-PG-30 Animal Naturals- K9 Puppy Gold- 30lb Container (FREE SHIPPING) $318.99 $277.52
AN-PG-4 Animal Naturals- K9 Puppy Gold- 4lb Container $58.49 $50.89
AN-PG-7 Animal Naturals- K9 Puppy Gold- 7lb Container $86.49 $75.25
Check the items you wish to purchase, then click


Features:

 
 
Trans Fat Free!

FOR

•  Newborn & Weaned Puppies
•  Pregnant & Nursing Mothers

PROMOTES

•  Antibodies & Resistance
•  Connective Tissue Strength
•  Joint & Soft Tissue Support
•  Bone Development
•  Healthy, Lean Growth

NEXT BEST THING TO MOM
Puppies grow healthiest and best on mother's milk. Science has decoded the immunity and growth secrets in canine milk to reveal over 1,000 bioactive properties. PUPPY
GOLD recreates canine mother's milk to promote immunity, stronger joints and lean growth.

“SWISS ARMY KNIFE” OF NUTRITION
PUPPY
GOLD contains canine milk's bioactive proteins, micellar casein, colostrum, glucosamine and more to give a giant head start in life. We even include canine milk's awesome immune factors, including lactoferrin. PUPPY GOLD helps produce not just healthier puppies, but healthier, longer living adults.

FOR MOM, TOO!
The same nutrients that help puppies after birth help them grow inside mom. Feeding PUPPY
GOLD to mothers helps producer healthier, more robust puppies and a stronger mom. Mothers look great during pregnancy & nursing and snap back fast afterwards .

NO MORE RUNTS
PUPPY
GOLD supplies every pup the pick of the litter's share of immune, joint and growth nutrients. Because they get their fair share, even small puppies can catch up and express their true (formerly hidden) genetic potential. No more runts!

IT'S THE GOLD STANDARD OF PUPPY FORMULAS!

Qusetions and Answers

WHAT IS PUPPY GOLD?
PUPPY
GOLD is a pure white powder mixed with food, or fed alone. PUPPY GOLD is the first formula patterned after the “gold standard” puppy formula, mother's milk. Turns out, canine milk is highly unique. PUPPY GOLD is designed to duplicate the many survival and growth advantages of mother's milk.

WHAT WILL PUPPY GOLD DO FOR MY PUPPY?
First, PUPPY
GOLD helps boost immunity. Second, tissue and protein synthesis is enhanced. Finally, maybe most important, joint and connective tissue formation is promoted. The combination of immunity, growth and structural strength produces rock solid bodies and health which can last a lifetime.

IS PUPPY GOLD BETTER THAN GOAT MILK?
Goat milk is the absolute perfect formula for…goats! Goat milk and canine milk are very different, because they're very different species. Dogs are mobile, smart predators and their milk reflects it. Goats are, well, goats. It's as absurd to feed goat milk to puppies as to feed cat milk to a baby whale. Mother's milk is species specific, and PUPPY
GOLD faithfully copies canine mother's milk without the use of trans fats .

HOW DOES PUPPY GOLD HELP BREEDING MOTHERS?
First, PUPPY
GOLD helps develop puppies inside the mother during pregnancy, sparing mom's muscle and bone mass. Second, during lactation nutrient needs rise 400%. Insert study She extracts this enhanced need from her own body stores of muscle and bone. Instead, PUPPY GOLD supplies milk synthesizing building blocks, and spares her body's own stores. The result is a shiny, robust and healthy mother during pregnancy and nursing.

HOW CAN IT IMPROVE MY BREEDING PROGRAM?
First, there are no runts. Every puppy gets ultimate nutrition. Second, breeders get a clearer picture of what dogs to breed. Often the pup that looks the best as an adult got the most milk supply as a pup. But, that may not be the best genetic animal (genotype), but the best nourished as a baby, and therefore, develops the most as an adult. (phenotype). PUPPY
GOLD ensures every pup gets an equal share of superrich milk. Call it the equalizer. When they grow up, you'll see which has the best genetics to breed. The true genotype value of an individual will be clearer.

DO THEY LIKE PUPPY GOLD?
Puppies and moms alike love PUPPY
GOLD. Natural milk protein taste and smell stimulates eating in all puppies. PUPPY GOLD is ideal for neonates to mature mothers.
 

HUMAN GRADE INGREDIENTS Partially predigested protein blend of zero lactose Casein, Lactoalbumin (Whey), Freeze dried colostrum, Micellar casein, Lactobacillus acidophilus/casei, latis, L-Arginine, L-Histidine, L-Glutamine, L-Taurine, Beef fat, Fractionated coconut oil (MCTs), Cold pressed safflower oil, DHA (docosahexaenoic acid omega-3 fatty acid, lactoferrin Glycerol, Predigested complex carbohydrate mix of medium and long chain glucose polymers, Resistant Starch, Modified tapioca starch, Glucosamine, Chondroitin sulfate, FOS, Calcium citrate, Calcium phosphate, Potassium chloride, Sodium, Chloride, Choline chloride, Ferrous sulfate, Inositol, Zinc sulfate, L-Carnitine, Niacinmide, Vitamin A palmitate, Thiamine HCL, Riboflavin, Pyridoxine, HCL, Manganese, Folic acid, Biotin, Sodium selenite, Vitamin D, Cyanocobalmin (B12), Digestive enzymes, Trace minerals, Chromium. Naturally preserved with Natural Vitamin E, C, Alpha and Beta carotene. With CANINE-LYTETM Electrolyte formula.

DIRECTIONS:

AS A MILK REPLACER
Prepare enough formula for use within 24-48 hours. Always refrigerate unused prepared formula.

Begin with boiled or bottled filtered water that has been allowed to cool to room temperature (70F).

To make one pint of formula, mix 1½ cups of water in a pint container/bottle. Add 2 scoops PUPPY GOLD powder. Mix/Shake until smooth.

FOR WEANING PUPPIES
Puppies should be weaned (removed from nursing) when they are about 25 days old.

Mix 1 scoop of PUPPY GOLD powder with every 1 cup of dog food. Add warm water (not to exceed 70F) as needed to produce a thick, soupy mixture.

FOR GROWING PUPPIES
Mix 1 scoop PUPPY GOLD with each 2 cups dog. Feed dry, or with water, or ad lib. Feed until 4-6 months old.

FOR PREGNANT AND NURSING BITCHES
PUPPY GOLD supplies nutrients to help puppies grow inside mothers, and provide milk. Sparing the mother's own muscle and bone mass helps moms quickly snap back to full health and vigor.

Provide one scoop PUPPY GOLD for every 10 lbs of body weight into daily food supply and continue one month (4 weeks) after weaning. Begin at start of conception and continue for two weeks after weaning.

Serving Size 1 Scoop (27 grams)
Servings Per Container 67

Protein.............. 31%
Fat……………...28%
Moisture…….…4%
Fiber……………3%
Ash……………..2%

 
 

STUDIES RELATING TO PUPPY GOLD NUTRIENTS AND FORMULATION

Am J Clin Nutr. 2004 Nov;80(5):1322-6. Related Articles, Links

Bone mass is recovered from lactation to postweaning in adolescent mothers with low calcium intakes.

Bezerra FF, Mendonca LM, Lobato EC, O'Brien KO, Donangelo CM.

Laboratorio de Bioquimica Nutricional e de Alimentos, Instituto de Quimica, Universidade Federal do Rio de Janeiro, Brazil.

BACKGROUND: Adolescent mothers may be at increased risk of irreversible bone loss during pregnancy and lactation, particularly when calcium intake is low. OBJECTIVE: Longitudinal changes in bone mass from lactation to postweaning were evaluated in 10 adolescent mothers aged 15-18 y who habitually consumed <500 mg Ca/d. DESIGN: Total-body bone mineral content (TBBMC), total-body bone mineral density (TBBMD), and lumbar spine bone mineral density (LSBMD) were measured at lactation (6-24 wk postpartum) and after weaning (12-30 mo postpartum). Serum hormones (intact parathyroid hormone, estradiol, and prolactin), serum calcium, and markers of bone turnover [urinary N-telopeptide cross-linking region of type I collagen (NTx) and plasma activity of bone alkaline phosphatase] were measured at lactation. RESULTS: TBBMC, total calcium content, TBBMD, and LSBMD increased from lactation to postweaning (P < 0.01). TBBMD and LSBMD were, respectively, 3.6% and 9.7% lower than predicted at lactation and 0.3% and 4.8% lower than predicted in the postweaning period. The increase in age-matched TBBMD adequacy was correlated with the time after resumption of menses (r = 0.86, P < 0.01). Calcium accretion from lactation to postweaning correlated negatively with estradiol (r = -0.86) and prolactin (r = -0.69) and positively with intact parathyroid hormone (r = 0.72) and NTx (r = 0.84) measured at lactation (P < 0.05). CONCLUSIONS: It appears that adolescent mothers with habitually low calcium intakes recover from lactation-associated bone loss after weaning. The rate of bone accretion, however, may not be sufficient to attain peak bone mass at maturity. Hormones regulating bone turnover during lactation may influence bone recovery in adolescent mothers.

Br J Nutr. 2005 May;93(5):593-9.

The influence of erythrocyte folate and serum vitamin B12 status on birth weight.

Relton CL, Pearce MS, Parker L.

Paediatric and Lifecourse Epidemiology Research Group, School of Clinical Medical Sciences (Child Health), Newcastle University, Sir James Spence Institute, Royal Victoria Infirmary, Newcastle upon Tyne NE1 4LP, UK. c.l.relton@ncl.ac.uk

The extent to which maternal folate and vitamin B12 modulate infant birth weight is unclear. The present study investigated mothers in early gestation (mean 11.5 (sd 5.8) weeks) and neonates, at delivery. Erythrocyte (RBC) folate (mothers: n 683; neonates: n 614) and vitamin B12 (mothers: n 534; neonates: n 614) were measured. Data on smoking habits were available for 44 % of pregnancies (n 443). The relationship between vitamin levels and birth weight standardized for gender and gestational age was investigated, using linear regression and adjusting for possible confounding variables (maternal age, parity). Results are presented as standardized regression coefficients (b). Increasing maternal age was associated with elevated RBC folate (b 0.11 (95 % CI 0.08, 0.15), P<0.001; n 674) and smoking was associated with a decrease in maternal RBC folate (b -1.38 (95 % CI -1.92, -0.86), P=0.001; n 319). Neonatal RBC folate was predicted by maternal RBC folate (b 0.08 (95 % CI 0.04, 0.11), P=0.001; n 315) and maternal vitamin B12 (b 0.08 (95 % CI 0.01, 0.16), P=0.02; n 252). Smoking influenced maternal vitamin B12 status (b -0.88 (95 % CI -1.49, -0.27), P=0.005; n 231). Using univariate regression, smoking significantly influenced infant birth weight (b -2.15 (95 % CI -3.24, -1.04), P<0.001; n 437). However, the effect of smoking on birth weight was statistically non-significant when considered in a multivariate regression model, leaving maternal RBC folate as the only significant predictor of birth weight (b 0.25 (95 % CI 0.08, 0.42), P=0.005; n 145). These findings suggest that maternal folate status is an important determinant of infant birth weight. The combined effects of smoking and reduced RBC status on birth weight require further investigation.

J Nutr. 2004 Jan;134(1):86-92. Related Articles, Links

Supplementation of sows with L-carnitine during pregnancy and lactation improves growth of the piglets during the suckling period through increased milk production.

Ramanau A, Kluge H, Spilke J, Eder K.

Institute of Nutritional Sciences, Martin-Luther University, Halle-Wittenberg, D-06108 Halle/Saale, Germany.

Recent studies showed that piglets of sows fed diets supplemented with L-carnitine grow faster during the suckling period than piglets of control sows fed diets without L-carnitine. This study was undertaken to investigate the effect of L-carnitine supplementation in sows on milk production and milk constituents. An experiment was performed in which two groups of 20 gilts each were fed diets with or without supplemental L-carnitine during pregnancy (0 vs. 125 mg L-carnitine daily/sow) and lactation (0 vs. 250 mg L-carnitine daily/sow). The experiment was continued over two reproductive cycles. L-carnitine-treated sows had larger litters (P<0.01) and higher litter weights (P<0.05) than control sows. Piglets of L-carnitine-treated sows had lower birth weights (P<0.05) but grew faster during the suckling period (P<0.01) and were heavier (P<0.05) at weaning than piglets of control sows. L-carnitine-treated sows had higher milk yields on d 11 and 18 of lactation than control sows (P<0.05). Milk of L-carnitine-treated sows had higher concentrations of total and free carnitine than milk of control sows (P<0.001); concentrations of fat, protein and lactose and the amounts of gross energy in the milk did not differ between the two groups of sows. The amounts of protein (P<0.05) and lactose (P<0.05) were higher in L-carnitine-treated sows than in control sows; the amount of energy secreted with the milk tended to be higher in carnitine-treated sows than in control sows (P<0.10). The study suggests that piglets of carnitine-treated sows grow faster during the suckling period than those of control sows because they ingest more nutrients and energy with the milk.

Antiviral Res. 2003 Nov;60(3):193-9. Related Articles, Links

Antiviral activity of lactoferrin against canine herpesvirus.

Tanaka T, Nakatani S, Xuan X, Kumura H, Igarashi I, Shimazaki K.

Dairy Science Laboratory, Graduate School of Agriculture, Hokkaido University, Sapporo, Hokkaido 060-8589, Japan. tetsuya@anim.agr.hokudai.ac.jp

Lactoferrin (LF) is an iron-binding protein that is found in milk and other mammalian secretions. We found that bovine lactoferrin (bLF) inhibited both the in vitro infection and replication of canine herpesvirus (CHV) in Madin-Darby canine kidney (MDCK) cells. Incubation of CHV with bLF prevented subsequent infection of MDCK cells. Furthermore, proteins from CHV-infected MDCK cells were resolved by SDS-PAGE, and then bLF CHV-binding proteins were identified by far Western blotting. We demonstrated that the anti-CHV activity of bLF was due to its interaction with CHV as well as with MDCK cells. Both the apo- and holo-forms of bLF inhibited virus multiplication independently of their iron-withholding properties. We also demonstrated that human LF had anti-CHV activity. Our findings suggest that LF could be effective in dogs to provide protection against CHV infection

Adv Exp Med Biol. 1994;357:71-90. Related Articles, Links

The effects of lactoferrin on gram-negative bacteria.

Ellison RT 3rd.

Department of Medicine, University of Massachusetts School of Medicine, Worcester 01655, USA.

Lactoferrin is an iron-binding protein found in human mucosal secretions as well as the specific granules of polymorphonuclear leukocytes. A variety of functions have been ascribed to the protein, and it appears to contribute to antimicrobial host defense. In particular, it has been shown to have direct effects on pathogenic microorganisms including bacteriostasis and the induction of microbial iron uptake systems. Still its overall physiologic role remains to be defined. It has appeared logical that antimicrobial activity of the protein arises from sequestration of environmental iron thereby causing nutritional deprivation in susceptible organisms. This argument is buttressed by the finding that selected highly virulent pathogens have evolved techniques to subvert this effect and use the protein as an iron source. However, recent observations indicate that the protein has additional properties that contribute to host defense. Work by several groups has shown that the protein synergistically interacts with immunoglobins, complement, and neutrophil cationic proteins against Gram-negative bacteria. Further, both the whole protein and a cationic N-terminus peptide fragment directly damage the outer membrane of Gram-negative bacteria suggesting a mechanism for the supplemental effects. This review will summarize these diverse observations with a consideration of how the in vitro work relates to the physiological role of the protein.

Br J Nutr. 2003 Nov;90(5):979-86. Related Articles, Links

Infant intake of fatty acids from human milk over the first year of lactation.

Mitoulas LR, Gurrin LC, Doherty DA, Sherriff JL, Hartmann PE.

Biochemistry and Molecular Biology, School of Biomedical and Chemical Sciences, Faculty of Life and Physical Sciences, The University of Western Australia, 35 Stirling Highway, Crawley WA 6009, Australia. Leon.Mitoulas@uwa.edu.au

Despite the importance of human milk fatty acids for infant growth and development, there are few reports describing infant intakes of individual fatty acids. We have measured volume, fat content and fatty acid composition of milk from each breast at each feed over a 24 h period to determine the mean daily amounts of each fatty acid delivered to the infant from breast milk at 1, 2, 4, 6, 9 and 12 months of lactation in five women. Daily (24 h) milk production was 336.60 (SEM 26.21) and 414.49 (SEM 28.39) ml and milk fat content was 36.06 (SEM 1.37) and 34.97 (SEM 1.50) g/l for left and right breasts respectively over the course of the first year of lactation. Fatty acid composition varied over the course of the day (mean CV 14.3 (SD 7.7) %), but did not follow a circadian rhythm. The proportions (g/100 g total fatty acids) of fatty acids differed significantly between mothers (P<0.05) and over the first year of lactation (P<0.05). However, amounts (g) of most fatty acids delivered to the infant over 24 h did not differ during the first year of lactation and only the amounts of 18:3n-3, 22:5n-3 and 22:6n-3 delivered differed between mothers (P<0.05). Mean amounts of 18:2n-6, 18:3n-3, 20:4n-6 and 22:6n-3 delivered to the infant per 24 h over the first year of lactation were 2.380 (SD 0.980), 0.194 (SD 0.074), 0.093 (SD 0.031) and 0.049 (SD 0.021) g respectively. These results suggest that variation in proportions of fatty acids may not translate to variation in the amount delivered and that milk production and fat content need to be considered.

Am J Clin Nutr. 2004 Jan;79(1):139-47. Related Articles, Links

Dose response of bone mass to dietary arachidonic acid in piglets fed cow milk-based formula.

Blanaru JL, Kohut JR, Fitzpatrick-Wong SC, Weiler HA.

Department of Human Nutritional Sciences, University of Manitoba, Winnipeg, Canada.

BACKGROUND: The addition of arachidonic acid (AA) and docosahexaenoic acid (DHA) to infant formula was recently approved in North America. In piglets, dietary AA is linked to elevations in bone mass. OBJECTIVE: The objective was to investigate the effects of varied amounts of dietary AA on bone modeling and bone mass with the use of the piglet model for infant nutrition. DESIGN: Male piglets (n = 32) were randomly assigned to receive 1 of 4 formulas supplemented with AA (0.30%, 0.45%, 0.60%, or 0.75% of fat) plus DHA (0.1% of fat) from days 5 to 20 of life. Measurements included biomarkers of bone modeling, fatty acid status, and whole-body and femur bone mineral content; bone area was measured by dual-energy X-ray absorptiometry. Differences among groups were detected with two-factor analysis of variance. Regression analyses were used to determine factors responsible for bone mineral content after dietary AA was accounted for. RESULTS: Proportions of AA in plasma, liver, and adipose were modified by the dietary treatments, but bone modeling was not affected. Liver AA was positively related to plasma insulin-like growth factor 1 and calcitriol and urinary N-telopeptide. Whole-body bone mineral content was elevated in the piglets fed 0.60% and 0.75% AA and was best predicted by dietary AA and bone resorption. CONCLUSIONS: This study confirms that dietary AA alters bone mass and clarifies the best amount of AA to add to the diet of pigs born at term. Because the amount of dietary DHA was held constant, whether other amounts of DHA are related to bone mass requires investigation.

Plasma beta-casomorphin-7 immunoreactive peptide increases after milk intake in newborn but not in adult dogs

M Singh, CL Rosen, KJ Chang and GG Haddad

Department of Pediatrics (Pulmonary Division), Columbia University College of Physicians and Surgeons, New York, New York 10032.

To determine the relation between milk intake and plasma levels of beta- casomorphins as a function of age, we studied the levels of beta- casomorphin-7 immunoreactive material (BCMIR) in 2- and 4-wk-old beagle neonates and adult dogs following milk intake. We used three kinds of milk: 1) bovine casein-based milk formula; 2) canine bitch's milk, and 3) soy protein-based (noncasein) formula. Using a new extraction technique, BCMIR was isolated from plasma and assayed by radioimmunoassay at 0, 2, 4, and 6 h after feeding. We found that BCMIR increased significantly in both 2- and 4-wk-old puppies post bovine and canine milk feeding, but not after soy protein formula. Base-line values were about 80-120 fmol/ml and increased to 100-120% 2 h after bovine casein intake in the older puppies and by 4 h in the younger group. BCMIR levels were undetectable in adult dogs before or after bovine milk feeding. Beta-casomorphin-7 added to plasma was rapidly degraded whereas BCMIR was stable. Chromatographic profiles demonstrated that the BCMIR moiety is larger than beta-casomorphin-7. Our data suggest that the appearance of BCMIR in the plasma of neonatal dogs is a result of beta-casein degradation along a permeable gastrointestinal mucosa. We speculate that morphiceptin and beta- casomorphins can be cleaved from BCMIR and may have important physiologic effects

Am J Vet Res. 1999 Sep;60(9):1088-91. Related Articles, Links

Comparison of insulin-like growth factor-I concentration in mammary secretions and serum of small- and giant-breed dogs.

White ME, Hathaway MR, Dayton WR, Henderson T, Lepine AJ.

Department of Animal Science, College of Agricultural, Food, and Environmental Sciences, University of Minnesota, St. Paul 55108, USA.

OBJECTIVE: To determine insulin-like growth factor-I (IGF-I) concentrations in canine mammary secretions and serum during lactation and to compare them between small and giant breeds of dogs. ANIMALS: 7 gestating Beagles and 4 gestating Great Danes. PROCEDURE: Dogs were fed a common nutritionally complete and adequate gestation and lactation diet. Milk samples were collected at postpartum hour 12 and postpartum days 3, 7, 14, 21, and 28 after IV oxytocin administration. Two puppies/litter were identified at whelping for collection of blood samples corresponding to the days of milk sample collection plus days 35 and 42. Maternal blood samples were obtained on days 1, 7, and 42 from Beagles and days 1, 7, and 28 from Great Danes and were acid/ethanol extracted and analyzed by use of a radioimmunoassay. RESULTS: Maternal serum IGF-I concentration was greater in Great Danes at all sample collection times. Similarly, colostrum from Great Danes contained more IGF-I, compared with that of Beagles (70 ng/ml vs 40 ng/ml, respectively). These values decreased to approximately 10 ng/ml by day 3 in both breeds and remained between 10 and 20 ng/ml for the duration of lactation. Growth rate and serum IGF-I concentration were greater in Great Dane puppies at birth to day 42. CONCLUSIONS AND CLINICAL RELEVANCE: High IGF-I concentration in colostrum may be biologically important for newborn puppies. Body mass and serum IGF-I concentration are directly correlated in growing Beagle and Great Dane puppies. Serum IGF-I concentration may be an indicator of growth potential in dogs.

Cornell Vet. 1993 Apr;83(2):107-16. Related Articles, Links

Serum chemistry and lipid profiles in neonatal beagle puppies fed homemade milk replacer formulas.

Chandler ML, Miller E, Olson PN, Ralston SL.

Department of Clinical Sciences, College of Veterinary Medicine, Fort Collins 80523.

Milk replacer formulas based on cow's milk and egg yolks are frequently recommended for use in neonatal puppies. These formulas are lower in protein, kilocalories, calcium, and phosphorus than bitch's milk. In addition, the cholesterol content is greater than bitch's milk. The effect of feeding these formulas on serum chemistry profiles, lipid profiles, and alkaline phosphatase isoenzyme profiles of 5-week-old beagle puppies was studied. Three groups of beagle puppies were fed bitch's milk (control) (n = 18), a homemade milk-egg-oil formula (Formula 1) (n = 18), or a homemade milk-egg-oil formula supplemented with additional calcium and phosphorous (Formula 2) (n = 18). Concentrations of serum urea nitrogen, albumin, and total CO2 were lower (P < 0.05), and concentrations of serum phosphorus, globulins, sodium, chloride, and cholesterol were higher (P < 0.05) in formula-fed puppies than bitch-fed puppies. Serum potassium concentration was lower in the puppies fed Formula 1 than in the control puppies (P < 0.05), and serum potassium concentration in the puppies fed Formula 2 was not significantly different from that in puppies fed Formula 1 or the control puppies. Total triglyceride (TG) and high density lipoprotein2 cholesterol (HDL2) concentrations were similar in all three groups of puppies but the combined high density lipoprotein1 (HDL1) plus low density lipoprotein (LDL) cholesterol fraction was higher (P < 0.05) in the formula-fed puppies and accounted for the majority of the increase in cholesterol. There were no differences (P < 0.05) in total serum alkaline phosphatase (ALP) or bone-derived ALP (BALP) concentrations among the groups, however there was a higher (P < 0.05) serum concentration of liver-derived ALP (LALP) in the Formula 1-fed puppies. Feeding homemade egg and cow's milk-based puppy replacement formulas is not recommended for long term use.

Probl Vet Med. 1992 Sep;4(3):545-50. Related Articles, Links

Feeding to optimize canine reproductive efficiency.

Moser E.

The relationship between nutrition and reproduction is a topic of importance to veterinarians and breeders. Nutrient deficiencies, excesses, and imbalances all are capable of altering reproductive performance. Healthy adult dogs should be fed a diet high in digestibility, low in residue, and high in nutrient bioavailability. Rarely is nutritional intervention necessary during the first half of gestation. During the last half of gestation, switching to a growth/lactation diet and increasing intake by 1.2 to 1.4 x maintenance is indicated. Depending on breed and pup numbers, at peak lactation, feed intake may need to be increased by 2 to 4 x maintenance. The feeding goals of lactation are to minimize loss of bitch body condition score and optimize milk production and pup growth. After weaning, the female who is in poor body condition should remain on a diet rich in protein, fat, and trace nutrients until she is physically/nutritionally repleted.

Am J Clin Nutr 2002 Mar;75(3):570-80

A randomized controlled trial of long-chain polyunsaturated fatty acid supplementation of formula in term infants after weaning at 6 wk of age.

Birch EE, Hoffman DR, Castaneda YS, Fawcett SL, Birch DG, Uauy RD.

Retina Foundation of the Southwest, Dallas, TX 75231, USA. ebirch@retinafoundation.org

BACKGROUND: The critical period during which the dietary supply of long-chain polyunsaturated fatty acids (LCPs) may influence the maturation of cortical function in term infants is unknown. OBJECTIVE: The aim of the present study was to determine the relative importance for maturation of the visual cortex of the dietary supply of LCPs during the first 6 wk of life compared with that during weeks 7-52. DESIGN: A randomized controlled clinical trial of LCP supplementation in 65 healthy term infants who were weaned from breast-feeding at 6 wk of age was conducted to determine whether the dietary supply of LCPs after weaning influenced the maturation of visual acuity and stereoacuity. RESULTS: Despite a dietary supply of LCPs from breast milk during the first 6 wk of life, infants who were weaned to formula that did not provide LCPs had significantly poorer visual acuity at 17, 26, and 52 wk of age and significantly poorer stereoacuity at 17 wk of age than did infants who were weaned to LCP-supplemented formula. Better acuity and stereoacuity at 17 wk was correlated with higher concentrations of docosahexaenoic acid in plasma. Better acuity at 52 wk was correlated with higher concentrations of docosahexaenoic acid in plasma and red blood cells. No significant effects of diet on growth were found. CONCLUSION: The results suggest that the critical period during which the dietary supply of LCPs can influence the maturation of cortical function extends beyond 6 wk of age.

Am J Vet Res. 2001 Aug;62(8):1266-72.

Related Articles, Links

Changes in protein and nutrient composition of milk throughout lactation in dogs.

Adkins Y, Lepine AJ, Lonnerdal B.

Department of Nutrition, College of Agricultural and Environmental Sciences, University of California, Davis 95616, USA.

OBJECTIVE: To evaluate changes in protein and nutrient composition of milk throughout lactation in dogs. SAMPLE POPULATION: Milk samples collected from 10 lactating Beagles. PROCEDURE: Milk samples were collected on days 1, 3, 7, 14, 21, 28, 35, and 42 after parturition and analyzed to determine concentrations of nitrogen, nonprotein nitrogen, casein, whey proteins, amino acids, lipids, lactose, citrate, minerals, and trace elements. Optimum conditions for separating casein from whey proteins and distribution of milk proteins throughout lactation were assessed by use of polyacrylamide gel electrophoresis. RESULTS: Protein concentration was high in samples collected on day 1 (143 g/L), decreased through day 21 (68.4 g/L), and increased thereafter. Concentration of nonprotein nitrogen did not change throughout lactation (5.7 to 9.9% of total nitrogen content). Casein-to-whey ratio was approximately 70:30 and remained constant throughout lactation. Lactose concentration increased from 16.6 g/L on day 1 to 34.0 to 40.2 g/L on days 7 to 42. Lipid concentration ranged from 112.5 to 1372 g/L. Citrate concentration increased from day 1 (4.8 mM) to day 7 (6.6 mM), then gradually decreased until day 42 (3.9 mM). Iron, zinc, copper, and magnesium concentrations decreased during lactation, whereas calcium and phosphorus concentrations increased. Calcium-to-phosphorus ratio remained constant throughout lactation (approx 1.6:1). Energy content of milk ranged from 1,444 to 1,831 kcal/L. CONCLUSIONS AND CLINICAL RELEVANCE: Protein and nutrient composition of milk changes throughout lactation in dogs. These data can provide valuable information for use in establishing nutrient requirements of puppies during the suckling period.

Hypertension. 2004 Jun 7 [Epub ahead of print] Related Articles, Links

Perinatal L-Arginine and Antioxidant Supplements Reduce Adult Blood Pressure in Spontaneously Hypertensive Rats.

Racasan S, Braam B, Van Der Giezen DM, Goldschmeding R, Boer P, Koomans HA, Joles JA.

Departments of Nephrology and Hypertension and Pathology, University Medical Center, Utrecht, The Netherlands.

Embryo cross-transplantation and cross-fostering between spontaneously hypertensive rats (SHR) and normotensive rats (WKY) suggest that perinatal environment modulates the genetically determined phenotype. In SHR the balance between NO and reactive oxygen species (ROS) is disturbed. We hypothesized that increasing NO and diminishing ROS in perinatal life would ameliorate hypertension in adult SHR. Pregnant SHR and WKY and their offspring received L-arginine plus antioxidants (vitamin C, vitamin E, and taurine) during the last 2 weeks of pregnancy and then until either 4 or 8 weeks after birth. Systolic blood pressure (SBP) and urinary excretion of protein, nitrates (NOx), and thiobarbituric acid reactive substances (TBARS) were measured. At 48 weeks of age rats were euthanized for glomerular counts. Perinatal supplements reduced SBP persistently in SHR and prevented the SBP increase observed in aging WKY. Initially NOx excretion was lower and TBARS excretion higher in SHR than WKY. There was a direct effect on NOx excretion in supplemented pregnant SHR and their offspring, but no increase was observed after stopping the supplements. TBARS excretion was only depressed up to 14 weeks by the supplements despite persistent differences in SBP. Consistent effects on nephron number were absent. Mild proteinuria, present in control SHR at 48 weeks, was prevented in all supplemented rats. Perinatal supplementation of NO substrate and antioxidants results in persistent reduction of SBP and renal protection in SHR, although effects on NOx and TBARS were only transient. This suggests a critical role for perinatal pro- and antioxidant balance in programming BP later in life.

 

 
 




Articles:

ANIMAL NATURALS
THE INSIDE TRUTH ON PET FOOD AND SUPPLEMENTS

  The pet food industry has some dirty secrets that they hoped you’d never discover...but I’m blowing the whistle on them. And are they gonna be mad.

  But you’re being ripped off and your animals are suffering for it, in my opinion. And it’s been going on for decades.

  Strong words, I know, but after 20 years studying what’s actually used in commercial pet foods these days, I just couldn’t keep my mouth shut any longer.

  That’s why I put together this special investigative report. Once you’ve “digested” all this information, I’m certain that you’ll be as disgusted and outraged as I am.

First things first (or what’s not in the food): Judging from their advertisements and pretty photos, commercial pet foods contain choice cuts of beef, fresh grains, chicken breast, meat, eggs, rice…all the nutrition your dog could ever need. The same wholesome quality you eat, right?

Wrong. Sounds great, I know, but look at it from a commercial point of view; those ingredients are costly, not to mention more desirable for human consumption. They’re just too expensive and good for lowly dogs.

  I’ve gotten down and dirty and checked out the commercial manufacturers and I can tell you (as can industry watchdogs) that none of the big commercial producers use any of the above desirable ingredients in their feeds. The pictures they use of wholesome foods are flat out lies.

Okay, so what is in the food? You might be sorry you asked. Remember your mental image and the photos they use of that wholesome healthy food, the beef, the chicken, those tasty whole grains…now, brace yourself for a reality check.

  Those choice cuts of beef are really cow brains, tongues, esophagi and fetal tissue that may be dangerously high in hormones. Much of the meat is diseased and even cancerous. Because using that great looking meat means they’d have to actually pay for it, and as you’ll learn, pet food companies don’t buy anything human-grade.

  Those “whole grains” have had the starch removed for cornstarch powder and often the oil has been extracted (by chemical processing) for corn oil, or are remnants from the milling process. Those grains that are truly whole have usually been deemed unfit for human consumption because of mold, contaminants, poor quality, or poor handling practices. Or worse, ultra-high pesticide concentrations.

  The last few years alone, dozens of dogs died from aflatoxin poisoning-traced to rotting grains rejected for human use, but just perfect for dog foods. After all, it’s only dogs, so who cares?

  Then, because the manufacturers know that the food is utterly devoid of any real nutrition it is “fortified” with vitamins and minerals. As sick as this fortification joke is, it’s made even worse by the fact that the nutrients added are also rejected for humans use and full of their own contaminants.

Why? Because the ingredients they are using are not wholesome, and the harsh manufacturing practices that are required to produce those cute shapes destroy what little nutritional value the food ever had. It’s kind of like depleted uranium—it looks the same but lacks the original zap.

Remember, everything in pet food is scrap material from the human
food industry. It’s unfit, unsafe or condemned for human use. Hello!

  So humans benefit all around at the expense of your pet. Pet food manufacturers get great prices on the “waste materials”. Hint—if pet food companies don’t buy it, it’s fed to pigs before slaughter. Human food processing companies save money twice, once when they sell the materials that to them are merely garbage, and a second time when they avoid having to pay to dispose of that same disgusting muck. Is that a great business or what?

Hungry yet?

It gets worse… If this sounds like some sort of conspiracy, that’s because it is, at least in my opinion. It’s not by chance that four of the five largest U.S. pet food companies are subsidiaries of the major multi-national food corporations: Heinz, Nestle, Mars, Colgate-Palmolive and Ralston-Purina (who disguises this by having two separate food companies, one for human and the other for animal feeds).

Remember the Recent E-Coli Scare? It’s no wonder that dogs everywhere aren’t dropping like flies from e-coli infections. It’s testimony to the hardy evolutionary design of our best friends that they can survive given the incredibly high degree of bacterial contamination in the commercial foods we give them. Where humans drop like flies with food poisoning, dogs tough it out; but they suffer in their own way, too.

  According to API, the Animal Protection Institute, the problem is extreme. Parts deemed unfit for human food, include animals classed as “4-D” (dead, diseased, dying or disabled). These animals (which do not meet human-food qualifications) are sent to pet food processors straight away.

  4-D meat includes meat that is revolting beyond words: pus-laden, gangrenous “green liver” syndrome and worse. Suffice to say, if you’ve ever seen the meat that goes into today’s pet foods, you would not feed it anymore.

And when you see the term “meat or poultry by-products” what does that mean? Quite simply, that’s what’s left of the carcass after human and mechanical de-boning and processing have been completed: bones, blood, pus, intestines, bowels, ligaments, subcutaneous fat, hooves, horns, beaks and any other parts that humans cannot or will not knowingly eat.

  Maybe by-products should be renamed “bye-bye products”. But because of their low cost, they’re a staple of modern-day pet food.

The fox is watching the doghouse! The Pet Food Institute (the trade association for pet food manufacturers) acknowledges the importance of by-products as a source of additional income for farmers and food processors.

  The truth is that most of these remnant foods are not only indigestible, they also provide a questionable source of nutrition for our animals. At best. So it strikes me as more than a little ironic that an organization such as this calls itself the Pet Food Institute. Perhaps what it should really be called is the “human food waste product recycling club”. At least that would be a more accurate description of their focus and intent. Call a spade a spade.

Remember the 70’s movie “Soylent Green”? In this cheesy movie, Charlton Heston screamed “Soylent Green is people!” Story line, they took people who died, ground them up, then fed them to other people in a product called Soylent Green. It was a well-guarded secret so nobody knew they were actually eating other people. Well modern day pet foods are the real life equivalent of “Soylent Green”.

  Fact is, the Soylent Green process feeds your dogs every day. Though you surely won’t find dogs or cats listed as ingredients on any pet food labels, the San Francisco Chronicle reported that euthanized companion animals were found in pet foods. This was vehemently denied by manufacturers; however, the American Veterinary Medical Association confirmed the Chronicle’s story.

  Think about it. Do you really believe the thousands of road kills, pets dying at veterinary clinics and animal shelters every day go to doggy heaven. I grew up with TV cartoons showing old racehorses going to the rendering glue factory. My research leads me to believe it’s the same for dogs today.

So how is this effecting my pet? “From his experience as a veterinarian and federal meat inspector, P. F. McGargle, D.V.M., has concluded that feeding slaughterhouse wastes to animals increases their chance of getting cancer and other degenerative diseases. Those wastes, he reports, can include moldy, rancid or spoiled meats as well as tissues severely riddled with cancer. These meat scraps can also contain hormone levels comparable to those that have produced cancer in laboratory animals.

  Dr. McGargle attributed these high levels to two causes: synthetic hormones routinely fed to livestock to stimulate rapid growth, and meat meal whose source is often glandular wastes and fetal tissues from pregnant cows. Both are naturally high in hormones. When livestock is slaughtered and the meat is processed, the hormones are still active. High hormone levels have the most severe effect on cats, which are extremely sensitive to them. The tissues or pellets that are used to fatten steers and caponize chickens, for example, are considered toxic to cats, even in very low levels. These hormones are not destroyed by the high temperature and pressure cooking that pet food manufacture requires.

And what in the world is that terrible smell? Ever walk around the back of a restaurant that has a fryer? Did you notice that awful greasy rancid smell? You know, the one near that dumpster labeled industrial grease or oil disposal?

  Ever feel like that freshly opened bag of dog food smelled exactly the same way? You should. It does smell the same. That’s because the fat added to most commercial dog foods is refined animal fat, recycled kitchen grease, and other oils too rancid or deemed inedible for human use.

  These rancid fats are generally sprayed directly onto dried kibble or extruded pellets to make a product which even a dog won’t eat more palatable. While certain fats provide the luster and sheen to your dogs coat, the sparkle to his eyes and the extra calories working dogs require, the rancid fats that are discarded by restaurants or are rendered from slaughterhouse animals are not nearly so beneficial.

  In fact, these rancid fats are being blamed for a host of new health related problems in dogs including; diarrhea, vomiting of bile, gas, bad breath and even severe digestive problems. In puppies and kittens, whose digestive tracts are not fully developed, these rancid fats can cause permanent damage leading to a lifetime of digestion and assimilation problems. Even in foods designed to be less allergenic, such as lamb and rice formulas, rancid fats can cause your pet to experience a reaction.

Do you put your dog “out to pasture”? Of course you don’t. Dogs don’t graze. And you don’t feed your dog wheat or hay or corn either. Or do you? In the last decade, as the populace of this country has moved its diet more and more towards grains and away from animal proteins, the amount of grain products in animal foods has increased proportionally.

  This has happened for two reasons; first, because we are consuming less meat, the amount of animals slaughtered has decreased and the byproducts of those slaughtered animals have undergone a like decline. Second, with an increase in human grain usage, grain unfit for human consumption, moldy grain and grain byproducts have all become progressively more available.

  It should come as no surprise that corn and wheat are far from ideal for dogs or cats from a nutritional standpoint. Cats in particular suffer from diets that include a predominance of grain. That is because cats are obligate omnivores. That is, they require meat to fulfill certain physiological needs.

  Another problem with feeding predominately grain based foods has to do with the grains themselves. Many of the grains that find there way into commercial pet foods have become wet, or moldy or contaminated by a fungus. In 1995 Nature’s Recipe was forced to pull nearly 20 million worth of dog food contained wheat contaminated by a fungus that produced vomitoxin and aflatoxin which is a kind of poison called a mycotoxin that is given off when mold grows.

  Peanuts are particularly susceptible to aflatoxin problems as the fungi which produce it tend to grow especially well on peanut hulls. Of course it probably won’t amaze you at this point to discover that peanut hulls are another major constituent of certain commercial pet foods.

  How could this terrible fungus occur in pet foods. Well…testing, which is not great even for human foods, is almost nonexistent for pet products.

Was that you or the dog? Flatulence. It’s funny to blame it on your dog when you do it, but not nearly so funny when your dog does it all the time. Nevertheless, soy, which has been known to cause flatulence in dogs, has become an increasingly common ingredient in commercial foods. Another increasingly common reason for canine farting is intestinal blockages.

  The culprit; among others, humectants – ingredients such as corn syrup and corn gluten meal which bind water molecules to prevent oxidation – also bind the water in such a way that the food actually sticks to the colon and may cause blockage and fermentation—methane gas. The blockage of the colon may well cause an increased risk of cancer of the colon or rectum.

Pickled or preserved? Sodium nitrite, butylated hydroxytoluene, ethoxyquin, butylated hydroxyanisole, propyl gallate, tertiary butylhydroquinone and other equally evil sounding compounds are being added to commercial dog foods.

  Why?

  Because when you combine all of the aforementioned toxic decomposed, diseased, hormone laden and rancid ingredients into one product, the only thing that will keep them from becoming so utterly putrid is to lace them with powerful chemicals to keep them “stable”.

  We might as well be storing them in formaldehyde. And if you think the names of these chemicals sound bad, take a look at the problems they cause; cancers of the stomach, intestines and blood, kidney damage, central nervous system disorders, depressed immune function, liver damage other forms of chronic and acute chemical toxicity.

  Even foods labeled “preservative free” may have one or more of the above chemicals added. That’s because the government does not require the manufacturer to list on the ingredient label anything added to ingredients prior to incorporation into the finished food. Thus, when rancid fat is preserved with ethoxyquin prior to arrival at a pet food manufacturers facility, the manufacturer, provided it does not add additional ethoxyquin to the material, is not required to list ethoxyquin anywhere on the label.

Just how prevalent are these preservatives in the food? A recent analysis determined that two-thirds of the pet foods produced in the US had synthetic preservatives added by the manufacturers. Of the remaining third, 90% included ingredients that were already stabilized by synthetic preservatives.

  Today, there are more than 8,600 recognized food additives with absolutely zero toxicity information on 45% of them! Of those that have been analyzed only 5% have had a complete health hazard assessment. Many of these chemicals are known to be carcinogenic above a certain level but are still allowed to be added in concentrations below the known carcinogenic amount.

  This is misleading because many of these compounds are not readily cleared from the body and can thus build up to very high levels over time.

  Ethoxyquin, for example, was found in dogs’ livers months after it had been totally removed from their diet.

Since when do dogs need to be fed insecticide as part of a healthy diet? The answer to this question should be obvious. NEVER! Here’s another alarming fact: 60% of all herbicides, 90% of all fungicides and 30% of all insecticides have been proven to cause cancer in and of themselves. Virtually all of these chemicals find there way into commercial pet food via the added grains and meats used.

  Worse still, upwards of 80% of these chemicals, 49,000 in all, have virtually no toxicity data. In other words neither you, nor I, nor any scientist in any lab in the world has any idea what they will do to you pet today, tomorrow or ten years from now.

  Another recent study found that these same chemicals lead to chromosomal mutations, double-strand DNA breaks and numerous mutations.

  To your dog, this means a greater incidence of cancer and other diseases. To your breeding program, this means lower survivability, mutated puppies and genetic aberrations that will make the pups non-salable.

  Just recently, I received the following notice from the FDA Center for Veterinary Medicine:

August 14, 1997

FDA REQUESTS THAT ETHOXYQUIN LEVELS BE REDUCED IN DOG FOODS In letters dated July 31, 1997, to manufacturers of ethoxyquin and trade associations for the pet food industry, FDA’s Center for Veterinary Medicine (CVM) requested that the maximum level for ethoxyquin in complete dog foods be voluntarily lowered to 75 parts
per million (ppm).

  Under the current food additive regulations, ethoxyquin is allowed at levels up to 150 ppm in complete dog foods (Title 21, Part 573.380 of the Code of Federal Regulations). However, after recently completing a scientific review of a voluntarily submitted study from the Monsanto Company, CVM has reason to believe that the 150 ppm level may not provide an adequate margin of safety in lactating female dogs and possibly puppies.

  The results from this study show that ethoxyquin levels above the current tolerance in dog foods produced no adverse reproductive effects. There was, however, an increase in a dark, reddish-brown pigment in the liver of female dogs immediately after completing a 6-week lactation. The liver pigment was identified as protoporphyria.

  During lactation, the female dogs consumed two to three times more food as a percentage of body weight than they did at maintenance, and this increased food consumption likely contributed to the increased pigment deposition in the liver and in the elevated serum enzymes. The increased pigment deposition and serum enzymes in lactating female dogs may be reversible when food consumption returns to maintenance, but it still constitutes a finding that must be further investigated.

  The Pet Food Institute has undertaken a study designed to show that ethoxyquin is an effective antioxidant at levels between 30 and 60 ppm in a complete dog food. FDA is closely monitoring the progress of this study. If new information becomes available that questions the safety of ethoxyquin at 75 ppm in dog food, or shows it to be an effective antioxidant at levels below 75 ppm, CVM will consider further action.

  Further information on this subject is available from FDA/CVM’s Division of Animal Feeds, 7500 Standish Place, HFV-220, Rockville, MD 20855 or by calling (301) 594-1724.

  I suggest you call the FDA at that phone number. After all, you own both the FDA and those phone lines because you pay for it all through your taxes. The FDA works for you, so start getting your money’s worth.

  What is particularly alarming about this document is the fact that the typically conservative FDA is making such a significant, almost radical recommendation. Generally, the data has to be overwhelmingly conclusive before they make such a strong statement.

  The second interesting element to the above is the fact that this report was submitted voluntarily by the Monsanto Corporation. Monsanto is the largest EQ manufacturer in the world. Results must have been alarming indeed for them to have allowed even this much significant data to reach the Feds.

  Of course, this on the heels of a final and even more alarming piece of information: Ethoxyquin cannot be detected once it has been added to a food source. This means you have absolutely no way of knowing if the pet food companies are complying or not. The number of foods to which EQ has been added, and the amount of EQ that has been put into those foods far exceeds initial approval guidelines.

  Monsanto and other ethoxyquin producing companies swear this chemical is safe. Of course, tobacco companies say the same for cigarettes.

  If Monsanto believes ethoxyquin is safe, then the CEO on down should begin eating it in the executive dining room, and feed it to their children. Come on fellas, walk your safety talk or take it off the market.

From the slaughterhouse floor to your dogs’ bowl…The making of a commercial pet food: When I was in college, one of the few professors I had who happened to have a sense of humor had a funny poster on his door. It read;

“The Real Scientific Method”

Step One: Get money to check out something you’re interested in.
Step Two: Take a guess about what you’re gonna find out.
Step Three: Check it out for a while and write something down.
Step Four: Determine what the stuff you wrote down might mean.
Step Five: Alter your guess to match your info, or alter your info to match your guess.
Step Six: Spend the next two years convincing people with good reputations to publish what you “discovered”.
Step Seven: Pedal your “discovery” to the highest bidder.
Step Eight: Make millions, retire to a “teaching post” where you can teach other idiots to do what you did.

  I always got a kick out of it when I came by his office. I don’t think it’s quite as humorous now, though. That’s because it’s far too close to reality to be that funny.

  When you think pet nutrition, you should be thinking pet food marketing.

  The first thing that commercial manufacturers do is assemble high-priced teams of marketing MBAs who spend millions to sell their products. They then create a diet working within specific guidelines, the main factor being bottom line profits. Want proof? A glance at any pet food industry related magazine reveals marketing articles out numbering nutritional ones by at least 10 to 1.

  Feeding trials are generally conducted from ten weeks to six months, however virtually no companies ever conduct long term or generational trials. Or safety tests, either.

  The most important step (as far as companies are concerned) is palatability testing. Basically, they want to make sure that dogs will eat more of their brand than comparable brand X. The same food chemistry wizards from these huge multinational companies that brought us Cheez Whiz, Tater Tots and Baco Bits open their trick bags of synthetic color and taste treats.

So using commercial pet food is basically like force feeding junk
food to your pet year-round.

  All pet foods, wet or dry undergo ultra high temperature processing. This is good from a bacterial standpoint, but nutritionally it’s devastating to an already deficient food. Most vitamins, amino acids and virtually all enzymes are damaged or destroyed by high temperature processing. In particular, the “cross linking” of amino acid bonds damages protein values.

  All in all, in my opinion, regardless of brand, commercial pet foods represent a clear and present danger to the health, appearance and fecundity of your animals.

Most supplements are even worse! In the attempt to fill the huge nutritional gaps and avoid the dangers of commercial dog food, you may try dietary supplement. After all, human dietary supplements have evolved from the snake oil image into super effective sports supplements-even into products like “Ensure” and other advertised on TV.

  But pet supplements are often made under conditions so bad and ingredients so contaminated they make pet food look positively healthy. Since no organization enforces purity or even the most basic of safety tests, many—if not most—pet supplements are dangerous to animals.

  This is partly due to the fact that pet supplements use the same diseased 4-D meat and dog food, but are not cooked or sterilized. Meaning the e. Coli, salmonella and other bad bugs are often at dangerous levels—many times over safety margins.

  I personally visited a supplement company located on a firewood lot in Reno Nevada. As wood lots go, this one was filthy. The product was made in an open barn invested with rats, mice and even bats that roosted in the eves. I won’t go into the sanitation of the mixer and other rumors swirling around its use. I understand he later moved to either Iowa or Idaho to continue his secretive ways with less overview.

So what in the world can I do about this?!? I wish the answer were simple. Unfortunately, that’s not the case. As a group we can lobby for improved standards of ingredients, control of additives and truth in labeling on all commercially prepared pet foods.

  The problem with this approach is two-fold. One, we can expect to pay substantially more for pet food if these regulations come to be; and second, it will take significant time for them to be enacted and enforced. All the while the health of our animals is suffering.

  The other option is to get a group of nutrition and biochemical Ph.Ds. together with a huge budget for research, and almost unlimited resources for human-grade nutrients like real roast chicken, etc.

  Unfortunately, neither of these is easy nor are they practical options.

  Perhaps the best thing that you can do is feed foods made by a new, cutting edge high-tech pet food producer that’s dragging pet food into the 21st century. The approach is radically simple—duplicate wild canine diets, then super fortify the products with the latest, most potent “nutriceuticals”.

  The thing that sets this company apart from all others is that it has innovated much of the most powerful and legitimate human natural nutrition over the last two decades.

  These high tech food supplements are designed to augment, and literally supercharge, any food you currently feed. These products are profoundly beneficial in two ways. First, they provide extra badly needed nutrition without the toxicity burden of conventional foods. Second, they unlock more nutrients from commercial foods you are forced to use.

There is hope. I predict that over the next few years, the same revolution of health knowledge that’s occurred in human nutrition will occur for pets. With new access to the information highway, this will occur quite fast. Starting now, the definition of health will change from a mere absence of disease to optimal functioning of all metabolic systems.

  As with tobacco companies, the public will become progressively aware of this violation of our trust. As that occurs, tougher standards and higher quality foods will come into existence. It’s about time.

  Leading the way will be this small high-end company that produces safe, healthy and effective products for dogs and cats today. Historically, due to cost of the product and limited funds for marketing, these companies have mainly sold there products to kennels, performance dog enthusiasts and professional trainers, who know all too well the problems associated with commercial foods and the benefits of a proper feeding program.

  ANIMAL NATURALS is the company I’m referring to. For nearly two decades this organization has been researching dietary strategies for human and canine athletes. Millions of human athletes, and now your pet, have benefited from these advances.

  ANIMAL NATURALS products have been born out of necessity. I hope that the contents of this report have enlightened you to the tragic abuse our animals are suffering at the hands of corporate America, and that you now have the knowledge and the desire to do something about it.

  Wishing you and your animals all the best in health and in life,

  Bob Fritz

Bob Fritz, President
ANIMAL NATURALS, LLC.

Check Out Here

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the news?

 

 

 





Additional Information:

The Commitment To a Healthy Planet, Healthy Pets

  As never before, consumers seek natural foods for themselves, their pets and to support the health of our planet. They recognize healthy people and pets depend upon a healthy earth. ANIMAL NATURALS is the preeminent pet line using human-grade nutrition with over 20 years success in natural nutrition. We are a “green” company that stresses recycling and conservation. ANIMAL NATURALS gives consumers the best in natural pet care while treating the earth with respect.

  1. Proven Effectiveness Two decades of unparalleled success in Olympics, pro sports, police dog and other grueling arenas prove our formulas the gold standard to which others are compared. Each strengthens metabolic weak links for a specific stage of life. New natural healing formulas rival prescription drugs.
  2. Natural Ingredients Beyond natural, beyond organic are “evofoods” that recreate evolutionary diets. In 1981, we originated evofoods and have improved them to a higher degree than anybody. Incredible smell and taste from the finest whole foods and extracts are unrivaled in the industry. No artificial colorings, flavorings or preservatives.
  3. Recyclable Packaging materials are blends of recycled and/or recyclable, biodegradable materials. Food ingredients are from sustainably grown, renewable stocks. We never knowingly use depleted, high conservation or endangered sources.
  4. State-of-the-Art Science For 20 years, we’ve created-then improved-the most advanced products in the world. Scientists help scan hundreds of medical journals for new breakthroughs in natural medicine, sports science, etc. to make products evermore powerful and cost effective.
  5. Clean Production We go the added step to produce under strict GMP standards. We believe you should not feed your pet any ingredient you would not eat yourself, or from a packaging facility you would not eat in.

WE MAKE NATURAL AWESOME

NATURAL USED TO MEAN LAST PLACE. When I began creating human and canine foods, natural was code for “ineffective”. Natural nutrition just wasn’t effective; natural athletes lost to druggies. Natural bodybuilders looked like scrawny kids next to druggies. In drug tested contests, performance levels fell dramatically.

To insiders, natural was a joke.

  22 years later, natural foods I helped develop make today’s human and canine athletes leaner, faster and stronger. Improvements in power, strength and endurance are almost beyond belief. These products allow athletes to train harder, more often. The day to day difference in accelerating recovery and reducing muscle damage exceed even performance drugs. As proof, natural athletes tested for drugs actually beat drugged athletes and shatter old drug records.

Natural is no longer a joke.
 
Literally millions of athletes have set new levels of
personal performance using these breakthroughs.

  How did these products grow to eclipse drugs and power virtually every modern athlete? Renowned scientists from local universities Stanford and Berkeley helped. So did pro athletes and dogs to test on. But the real key to ongoing innovation of these natural products was not science, but Nature. Unlocking more hidden genetic potential required a fresh look at the evolutionary design of humans and canines.

  By coaxing Nature’s DNA secrets from the human, canine and equine body, my past products have changed the rules for drug-free performance. But my newest products surpass everything that’s gone before, in my opinion. These formulas contain power and safety impossible just a few years ago. Since increased performance is founded on health, overall health is raised.

  And my new therapeutics make natural healing a reality. Until now, natural remedies for pets were even more of a joke than nutrition used to be. A nice idea that just did not work.

My new healing formulas rival prescription drugs
effectiveness, yet are 100% natural…and cost less!

  22 years ago, I transformed natural from a joke into a revolution, changing sports forever. It’s hard to find a serious athlete who has not raised performance and health through the use of these products. Everyday, throughout the world, Professional, Olympic and other athletes use these formulas to compete against—and beat—drugged athletes.

  Below is a partial list of these landmark products.

PRODUCT DEVELOPMENT
UNIPRO: Marketing Manager, November 1982 to January 1987.

1982

  • AMINO 1000™ First sports nutrition application of milk protein peptide-free
    form amino acids mixtures.
  • Created Professional and Olympic Athlete Research Program.

1983

  • CARBOPLEX™ First sports nutrition application of glucose polymers.
  • LIPOPLEX™ First sports nutrition application of MCTs (medium-chain triglycerides).
  • OXYGIZER™ First American sports nutrition application of alpha ketoglutarate
    (PAK: Pyridoxal 5 phosphate alpha ketoglutarate).

1984

  • BCAAS™ First American sports nutrition application of branched-chain
    amino acids (L-Leucine, L-Isoleucine, L-Valine)
  • CREATINE 1500™ First American sports nutrition application of creatine.

1985

  • JOURNAL OF SPORTS SCIENCE First sports science abstract review for non-scientists.
  • First sports nutrition advisory board of multi-disciplinary front-line researchers.
  • First American research study of PAK: co-design of experimental protocol.

1986

  • High school football workout book with Howie Long and Lyle Alzado, 39 pg.
  • PAK study presented at ACSM (American College of Sports Medicine).

CHAMPION NUTRITION: Director of Sales and Marketing,
January 1987 to January 1990

1987

  • MUSCLE N2ITRO™ First American sports nutrition succinate application;
    Point of sale literature, print ad.

1988

  • POWER PASTA™ First sports nutrition application of protein/carbohydrate
    enriched pasta. Label, point of sale literature, print ad.
  • METABOLOL II™ First L-lactate enhanced Optimizer: research, label and ad copy.
  • CYTOMAX™ STUDY # 1 Co-design of experimental protocol.
  • METABOLOL II STUDY # 1 Co-design of experimental protocol.

1989

  • CYTOMAX STUDY # 2 Co-design of experimental protocol.
  • METABOLOL II STUDY # 2 Co-design of experimental protocol.
  • CYTOMAX™ First sports nutrition application of concentrated L-lactates.
  • CYTOMAX STUDY # 1 Presented study at International Sports Congress,
    Leningrad, USSR via Dr. Thomas Fahey.

ABR/APRL: President, January 1990 to 2000.

1990

  • METABOLOL II STUDY # 1 Presented study at ACSM via Dr. Fahey.
  • METABOLOL II STUDY # 2 Presented study at ACSM via Dr. Fahey.
  • PEAK PERFORMANCE™ First commercial canine sports nutrition supplement.
  • STEROIDS AND DOGS HANDBOOK Academic review, 50 pg.

1991

  • HOME ANABOLIC KIT™ First sports nutrition application of Nitrogen
    Balance testing. FDA approved, patented.
  • MUSCLE CHEM™ Advanced Optimizer.
  • MAXIMIZE™ Advanced Amino Acid.
  • STEROID ALTERNATIVE HANDBOOK Co-author with Dr. Thomas Fahey, 197 pg.

1992

  • WEIDER HOME ANABOLIC KIT Second generation.
  • PRE FLEX HYPERDRIVE™ First effervescent version of L-lactate supplement.
  • N2ITRO FIRE™ Nitrogen balance optimizing powder.
  • POST FLEX PM™ Pre-sleep anti-catabolic formula.
  • HOME ANABOLIC KIT STUDY #1.

1993

  • ELITE ATHLETE TESTING SERVICE First mail-in steroid hormone lab testing.
  • Developed 7 Products for GEN LINC.

1994

  • POWER JERKY™ First USDA approved, all natural nutritionally fortified jerky for athletes.
  • PEAK CONDITION Sports supplement for dogs.

1995

  • CORT BUSTER™ First commercial sports nutrition application of
    phosphatidylserine as oral cortisol antagonist. Patent pending.

1996

  • PEAK GROW™ First commercial reproduction of canine bitch milk.

1997

  • POWER MEAT™ First commercial mixture of USDA human grade wild meats and PEAK 2.
  • PEAK STRIDE™ Joint mobility canine enhancer.
  • PEAK SENIOR™ Anti-aging canine formula.
  • FAST TWITCH™ Sprint dog (greyhound) formula.

1998

  • PEAK EQUINE™ Race horse supplement.
  • PERFORMANCE DOG MAGAZINE Magazine devoted to performance dogs.
  • MUSCLE MILK™ First human bodybuilding product based on human milk profile.
  • ENDOCRE3™ First endogenous creatine formula with glycocyamine.

ANIMAL NATURALS, LLC CEO 2002 to Present.

  • MUSCLEJACK™ First commercial use of Longifolia.
  • EVOBODYBUILDING™ Natural diet and workouts based on the
    inner cavemen in modern humans, 116 pages.
  • CARNIVORE COOKIES TM Fortified gourmet “superbiscuit” using
    Rhodiola and other functional foods.
  • FEEL FULL™ Appetite suppressant using nutrients to raise body’s own feel full hormone, CCK.
  • SHOW & GO™ Performance and appearance natural equine product.
  • JOINT RE-GEN™ First human commercial activated glucosamine topical spray.
  • COOL SPOT™ First 100% natural commercial spray to guarantee total recovery from hot spots.

NEW PRODUCT CATEGORIES HELPED CREATE

  1. Amino Acids (Whey peptide/casein free-form).
  2. BCAAs (Branched-chain amino acids).
  3. Complex Carbohydrate Powders (CARBOPLEX, etc).
  4. MCTs.
  5. Alpha KetoGlutarates.
  6. L-lactate Polymers (CYTOMAX, etc).
  7. Home Anabolic Kits.
  8. Defined Predigested Meals for Dogs.
  9. Mail-In Hormone Tests.
  10. Sport Jerky (POWER JERKY, etc).
  11. Drug-Free Cortisol Antagonists.
  12. Glucose Polymer/Amino Acid Fortified Pasta.
  13. Human Grade Canine Metabolic Optimizers.
  14. Human Breast Milk Analogs (MUSCLE MILK).
  15. Endogenous Creatine Synthesizer (ENDOCREATINE).
  16. Freeze dried raw meat with daily RDAs (POWERMEAT).
  17. Super fortified gourmet meat dog biscuits (CARNIVORE COOKIES)
  18. Longifolia testosterone prohormone. (MUSCLEJACK).
  19. Topical hyaluronate spray (JOINT RE-GEN).

WRITING

  • 50 + National articles.
  • 7 Newsletters.
  • Publisher, PERFORMANCE DOG
  • 50 + Brochures.
  • 75 + National print ads.
  • 3 radio ads.
  • 4 Books.
  • 1 Infomercial, LEAN SOLUTION.

PATENTS

  • 2 issued.
  • 3 pending.



Average Customer Review: Based on 2 Reviews. Write a review.

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  1 of 1 people found the following review helpful:
 
love it!! June 11, 2008
Reviewer: samantha faryna from ewing, NJ United States  
our Golden Retriever pup love this stuff!  I even give a little bit to my German Shepherd as a treat in her food....

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  2 of 2 people found the following review helpful:
 
Best Puppy Supplement we have used July 5, 2006
Reviewer: Bill from North Carolina  
This is a great product for puppies and bitches.  Highly recommend!!!!

Was this review helpful to you?


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